Capecitabine, DNA/RNA Synthesis Inhibitor

Capecitabine is an oral proagent that is converted to its active metabolite, 5-FU, by thymidine phosphorylase. - Mechanism of Action & Protocol.

Gupta Cancer Part 2 Flashcards

5-Fluorouracil (5-FU) resistance and the new strategy to enhance the sensitivity against cancer: Implication of DNA repair inhibition - ScienceDirect

Interference with Nucleic Acid Synthesis - Creative Biolabs

Mechanism of action of capecitabine upon conversion into active 5-FU.

is a tumor-selective fluoropyrimidine carbamate, which achieves higher intratumoral 5-FU level with lower toxicity than 5-FU.

Capecitabine

DNA Synthesis Inhibitors- Definition, Examples, Inhibition, Resistance

Nucleoside analogues and nucleobases in cancer treatment - The Lancet Oncology

Capecitabine-d11, Stable Isotope

Full article: DNA synthesis inhibitors for the treatment of gastrointestinal cancer

5‑Fluorouracil and capecitabine therapies for the treatment of colorectal cancer (Review)

TAS1553, a small molecule subunit interaction inhibitor of ribonucleotide reductase, exhibits antitumor activity by causing DNA replication stress